Browsing by Subject "air pollution"

Now showing 1 - 6 of 6
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    Estimated validity and reliability of on-board diagnostics for older vehicles: comparison with remote sensing observations
    (2011-09) Supnithadnaporn, Anupit; Noonan, Douglas S.; Samoylov, Alexander; Rodgers, Michael O.
    Based on requirements under the Clean Air Act Amendments of 1990, most state vehicle inspection and maintenance (I/M) programs have, since 2002, replaced the tailpipe emission testing with the on-board diagnostic (OBD) II testing for 1996 model and newer vehicles. This test relies on the OBD II system to give the pass or fail result, depending on certain conditions that might cause the vehicle to emit pollution 1.5 times higher than the regulated standard. The OBD II system is a computer and sensors installed in the vehicle to monitor the emission control units and signal if there is any malfunction. As a vehicle ages, its engine, pollution control units, and OBD II system deteriorate. Because the OBD II system's durability directly influences the test outcome, it is important to examine the fleetwide trend in the OBD II test results in comparison with an alternative measure of identifying high emitting vehicles. This study investigates whether the validity and reliability of the OBD II test is related to the age of the OBD II system installed in the fleet. Using Atlanta's I/M testing records and remote sensing device (RSD) data collected during 2002-2005, this research establishes the convergent validity and interobserver reliability criteria for the OBD II test based on on-road emissions measured by RSDs. The study results show that older vehicles exhibit significantly lower RSD-OBD II outcome agreement than newer vehicles. This suggests that the validity and reliability of the OBD II test may decline in the older vehicle fleets. Explanations and possible confounding factors for these findings are discussed.
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    Residential Proximity to Traffic-Related Pollution and Atherosclerosis in 4 Vascular Beds Among African-American Adults: Results From the Jackson Heart Study
    (Oxford Academic, 2016-11-15) Wang, Yi; Wellenius, Gregory A.; Hickson, DeMarc A.; Gjelsvik, Annie; Eaton, Charles B.; Wyatt, Sharon B.; Environmental Health Science, School of Public Health
    To our knowledge, no study has investigated the association of long-term exposure to traffic pollution with markers of atherosclerosis in 4 vascular beds simultaneously in an all-African-American cohort. Among participants in the Jackson Heart Study (Jackson, Mississippi; baseline mean age = 55.5 (standard deviation, 12.7) years), we used linear regression to estimate percent differences in carotid intima-media thickness (CIMT) at baseline (2004) and used modified Poisson regression (robust error variance) to estimate prevalence ratios for peripheral artery disease (PAD), coronary artery calcification (CAC), and abdominal aortic calcification (AAC) at the first follow-up visit (2005–2008) for persons living less than 150 m (versus more than 300 m) from major roadways, adjusting for confounders. Living less than 150 m from such roadways was associated with a significant 6.67% (95% confidence interval: 1.28, 12.35) increase in CIMT (4,800 participants). PAD prevalence among persons living less than 150 m from a major roadway was 1.17 (95% confidence interval: 0.73, 1.86) times that of persons living more than 300 m away (4,443 participants), but this result was not statistically significant. There was no association for CAC or AAC. The association with CIMT was stronger in participants with a cardiovascular disease history than in those without one (P = 0.04). We observed an association in the carotid vascular beds but not the coronary, abdominal, or peripheral vascular beds. Our results highlight the need to consider residential proximity to roadways as a potential cardiovascular disease risk factor for blacks.
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    Short-term Changes in Ambient Particulate Matter and Risk of Stroke: A Systematic Review and Meta-analysis
    (2014-08) Wang, Yi; Eliot, Melissa N.; Wellenius, Gregory A.
    Background Stroke is a leading cause of death and long‐term disability in the United States. There is a well‐documented association between ambient particulate matter air pollution (PM) and cardiovascular disease morbidity and mortality. Given the pathophysiologic mechanisms of these effects, short‐term elevations in PM may also increase the risk of ischemic and/or hemorrhagic stroke morbidity and mortality, but the evidence has not been systematically reviewed. Methods and Results We provide a comprehensive review of all observational human studies (January 1966 to January 2014) on the association between short‐term changes in ambient PM levels and cerebrovascular events. We also performed meta‐analyses to evaluate the evidence for an association between each PM size fraction (PM2.5, PM10, PM2.5‐10) and each outcome (total cerebrovascular disease, ischemic stroke/transient ischemic attack, hemorrhagic stroke) separately for mortality and hospital admission. We used a random‐effects model to estimate the summary percent change in relative risk of the outcome per 10‐μg/m3 increase in PM. Conclusions We found that PM2.5 and PM10 are associated with a 1.4% (95% CI 0.9% to 1.9%) and 0.5% (95% CI 0.3% to 0.7%) higher total cerebrovascular disease mortality, respectively, with evidence of inconsistent, nonsignificant associations for hospital admission for total cerebrovascular disease or ischemic or hemorrhagic stroke. Current limited evidence does not suggest an association between PM2.5‐10 and cerebrovascular mortality or morbidity. We discuss the potential sources of variability in results across studies, highlight some observations, and identify gaps in literature and make recommendations for future studies.
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    Substantial Decreases in U.S. Cities’ Ground-Based NO2 Concentrations during COVID-19 from Reduced Transportation
    (MDPI, 2021) Heintzelman, Asrah; Filippelli, Gabriel; Lulla, Vijay; Earth Sciences, School of Science
    A substantial reduction in global transport and industrial processes stemming from the novel SARS-CoV-2 coronavirus and subsequent pandemic resulted in sharp declines in emissions, including for NO2. This has implications for human health, given the role that this gas plays in pulmonary disease and the findings that past exposure to air pollutants has been linked to the most adverse outcomes from COVID-19 disease, likely via various co-morbidities. To explore how much COVID-19 shutdown policies impacted urban air quality, we examined ground-based NO2 sensor data from 11 U.S. cities from a two-month window (March–April) during shutdown in 2020, controlling for natural seasonal variability by using average changes in NO2 over the previous five years for these cities. Levels of NO2 and VMT reduction in March and April compared to January 2020 ranged between 11–65% and 11–89%, consistent with a sharp drop in vehicular traffic from shutdown-related travel restrictions. To explore this link closely, we gathered detailed traffic count data in one city—Indianapolis, Indiana—and found a strong correlation (0.90) between traffic counts/classification and vehicle miles travelled, a moderate correlation (0.54) between NO2 and traffic related data, and an average reduction of 1.11 ppb of NO2 linked to vehicular data. This finding indicates that targeted reduction in pollutants like NO2 can be made by manipulating traffic patterns, thus potentially leading to more population-level health resilience in the future.
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    Taking a stand against air pollution – the impact on cardiovascular disease: A Joint Opinion from the World Heart Federation, American College of Cardiology, American Heart Association, and the European Society of Cardiology
    (Oxford, 2021-01) Brauer, Michael; Casadei, Barbara; Harrington, Robert A.; Kovacs, Richard; Sliwa, Karen; Medicine, School of Medicine
    Although the attention of the world and the global health community specifically is deservedly focused on the COVID-19 pandemic, other determinants of health continue to have large impacts and may also interact with COVID-19. Air pollution is one crucial example. Established evidence from other respiratory viruses and emerging evidence for COVID-19 specifically indicates that air pollution alters respiratory defense mechanisms leading to worsened infection severity. Air pollution also contributes to co-morbidities that are known to worsen outcomes amongst those infected with COVID-19, and air pollution may also enhance infection transmission due to its impact on more frequent coughing. Yet despite the massive disruption of the COVID-19 pandemic, there are reasons for optimism: broad societal lockdowns have shown us a glimpse of what a future with strong air pollution measures could yield. Thus, the urgency to combat air pollution is not diminished, but instead heightened in the context of the pandemic.
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    Taking a Stand Against Air Pollution—The Impact on Cardiovascular Disease
    (Elsevier, 2021) Brauer, Michael; Casadei, Barbara; Harrington, Robert A.; Kovacs, Richard; Sliwa, Karen; Medicine, School of Medicine
    Although the attention of the world and the global health community specifically is deservedly focused on the COVID-19 pandemic, other determinants of health continue to have large impacts and may also interact with COVID-19. Air pollution is one crucial example. Established evidence from other respiratory viruses and emerging evidence for COVID-19 specifically indicates that air pollution alters respiratory defense mechanisms leading to worsened infection severity. Air pollution also contributes to co-morbidities that are known to worsen outcomes amongst those infected with COVID-19, and air pollution may also enhance infection transmission due to its impact on more frequent coughing. Yet despite the massive disruption of the COVID-19 pandemic, there are reasons for optimism: broad societal lockdowns have shown us a glimpse of what a future with strong air pollution measures could yield. Thus, the urgency to combat air pollution is not diminished, but instead heightened in the context of the pandemic.